1. Drug-induced hepatitis
Characterized as follows: ① past history with drugs known to have a variety of drugs can cause varying degrees of liver damage, such as isoniazid, rifampicin can cause similar clinical manifestations and viral hepatitis; pairs of long-term use vinegar Ding phenol, methyl Toba, etc. can cause chronic active hepatitis; chlorpromazine, methyl testosterone, arsenic, antimony agent, ketoconazole, etc. can cause cholestatic hepatitis; ② clinical symptoms of light, single ALT increased eosinophils increased; ③ good ALT after stopping the symptoms gradually returned to normal.
2. Cholelithiasis
There Biliary Colic past history of fever chills, right upper quadrant pain, Are sign (Murphy sign)-positive, WBC increased neutrophils increased.
3. Primary biliary cirrhosis France
① middle-aged women, characterized by more common; ② significant jaundice, skin itching, often xanthoma, hepatosplenomegaly obviously, ALP increased significantly in the majority of anti-mitochondrial antibody-positive; ③ mild liver damage; ④ HBV negative things.
4. Hepatolenticular degeneration (Wilson disease)
Often family history, multiple manifestations of thick limb tremor, increased muscle tone, corneal edge of the green, brown pigment ring (KF ring), blood copper and plasma ceruloplasmin lower urinary copper increased, while the slow living liver blood copper and copper Blue protein increased significantly.
5. Acute fatty liver of pregnancy
Mostly occurs in late pregnancy. Clinical features are: ① the early onset of acute severe upper abdominal pain, amylase increased, it may be acute pancreatitis; ② Notwithstanding the heavy jaundice, serum direct bilirubin increased, urine bilirubin, but often negative. Domestic reporting this phenomenon is also seen in acute fulminant hepatitis, for reference; ③ frequently used in liver failure that is, before the advent of severe bleeding and renal function impairment, ALT increased, but the musk deer often turbid normal; ④ B-mode ultrasound examination for fatty liver waveform to facilitate early diagnosis, confirmed by pathological examination. Pathological features of the hepatic lobule to the increase in band cells, the cytoplasm filled with fat vacuoles without large liver cell necrosis.
6. Extrahepatic obstructive jaundice
Such as pancreatic cancer, total bile duct cancer, chronic pancreatitis and other required identification.
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